Fatty liver refers to fatty liver (primarily triglycerides) in which excess fat accumulates in liver cells, with a marked increase in the fat drop in liver cells, and is typically imaged as having a fat content in the liver greater than 5% of the liver weight. According to a history of non-alcoholic fatty liver and non-alcoholic fatty liver, which are closely related to obesity, diabetes, hyperlipidemia and other metabolic disorders, are currently the most common types of liver disease. Many people do not think of fatty liver as 'important', in fact, fatty liver can gradually progress to fatty liver disease, liver fibrosis, cirrhosis, and even liver cancer.
Non-alcoholic fatty liver disease is closely related to the 'metabolic syndrome'. High-calorie, high-fat, high-sugar diets lead to excess energy, obesity, especially abdominal obesity, are central risk factors; insulin resistance causes liver lipid metabolism disorder, fat accumulation in the liver; the incidence of diabetes, hyperlipidemia, and hypertension is significantly elevated. In addition, long-term sedation, sleep deprivation, and genetic factors are also involved.
Alcoholic fatty liver is caused by direct liver damage caused by long-term alcohol consumption. Some patients may experience weakness, upper right abdominal discomfort or aches, decreased appetite, swelling, and other non-specific symptoms. As the disease progresses to fatty hepatitis, liver cell damage increases, and transaminase increases; further development of hepatic fibrillation is not typical, and symptoms are not severe until the stage of hepatic cirrhosis, jaundice, digestive bleeding, and so on.
A small number of patients can skip the stage of hepatic cirrhosis and progress directly to liver cancer. Blood samples test liver function (transaminases), blood lipids, blood sugar, uric acid, etc. to assess metabolic status and degree of liver damage.
In patients with suspected progression, instantaneous elastic imaging of the liver can be used to assess the degree of hepatic fibrosis, when necessary to make a precise diagnosis. Fatty liver is currently free of pharmaceuticals and lifestyle intervention is the most fundamental treatment measure. Weight loss is at the core of treatment.
Clinical studies have confirmed that 5%-10% weight loss can significantly improve liver fat change and inflammation, and weight loss targets are typically 7%-10% of weight; Adjusting dietary structure, reducing refined sugar and saturated fat intake, increasing dietary fiber, controlling total calories; adhering to regular aerobic and resistance exercises, at least 150 minutes of moderate intensity exercise per week; strictly abstaining from alcohol. For people with synthetic diabetes, hyperlipidemia, blood glucose and lipid need to be actively controlled. Some patients may use anti-hepatitis medications such as acne lipid eczema, hydrophizines, etc.
as supportive therapies under the direction of a doctor. Fatty liver is reversible and the earlier intervention benefits the greater. Fatty liver should be found to change lifestyle and should be regularly monitored, checked for liver function and evaluated for disease progression.
If there has been a sustained elevation of transaminase or signs of hepatic fibrosis, it should be diagnosed according to the medical specification. Hold your mouth, move your legs, control your weight, and the vast majority of patients with fatty liver can return to normal.
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