Gout is a chronic manifestation of gout. Gout is found in many parts of the body, most commonly around the first joints, fingers, ears, elbows, and elbows, with yellowish-white or skin-colored joints, hardness, and gradual growth. Gout causes recurrent seizures, difficult to digest, erodible joints and bones, causing wrinkles, white uric acid crystals after rupture, and is not easily cured, severely affecting joint function and aesthetics.
The appearance of gout stones often suggests that gout has entered a chronic period, with a significant increase in long-term uric acid. The formation of gout stones is closely related to long-term persistent high uric acid haemorrhage. When the length of the uric acid exceeds the concentration of sodium (about 420 μmol/L, at body temperature and pH), the uric acid crystals are deposited in the lining of the joints, cartilage, musculoskeletal and soft tissues, and gradually form gout stones.
The core mechanism of gout stones is: repeated uric acid crystalline deposition activates giant cells and inflammatory responses, surrounding the formation of fibrous membrane envelopes, gradually enlarging and enlarging. High risk factors include: long-term significantly elevated blood uric acid levels (especially above 540 μmol/L), recurrent acute seizures of gout, long-term disease, dysfunction of the kidneys leading to decreased urine excretion, use of diuretics, obesity, alcohol, etc. Once a gout is formed, it often takes longer to dissolve, even if the blood uric acid is normal.
Clinical manifestations of gout are subcutaneous lumps, gross hardness, white activity or viscosity to deep tissue, normal or thin skin surface, redness, breakage of a pencil or toothpaste-like substance (uric acid crystallization) and short-term wound healing. Gastric stones are most commonly found in the first pair of joints (thigh ankles), but can also affect the fingers, wrists, elbows, knees, ankles, and soles, resulting in joint swelling, pain, and limited activity. When gastric stones erode the cartilage and bones of the joints, the imaging can be seen as 'slippery' bone damage, and the joints can deform.
Joint ultrasound can show 'double trajectory signals' of urine nitrate crystalline deposition formation, and dual-energy CT can be invariant. It clearly shows the distribution and content of urine nitrate crystals and is an important tool for assessing the urethral stone load. Urine nitrate crystals can be detected by penetrating or breaking urine secretions.
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